Hyperbaric oxygen therapy offers support:
Hyperbaric oxygen therapy (HBOT) has been shown in some studies to help combat degeneration by contributing to the regeneration of tissue and blood vessels.
The therapy stimulates the development of new blood vessels in areas of the body where circulation is compromised and is utilized by some physicians to help treat Coronary Heart Disease, Macular Degeneration, Parkinson’s disease, Alzheimer’s disease, Arthritis and immune-related diseases.
HBOT also helps promote collagen activation to help battle the signs of aging including skin damage and elasticity.
Clinical studies have demonstrated the benefits of HBOT for age-related degenerative conditions by providing cellular aid to all organs in the body to promote health and beauty.
BDNF – Brain-derived neurotrophic factor – is a protein the body releases to stimulate the production of new brain cells. A process is known as neurogenesis. You need this protein to support your learning, memory, higher thinking, and other similar brain functions. Of important interest, the protein is found to be significantly reduced in the brains of people with Alzheimer’s, Parkinson’s, and Huntington’s disease. Researchers are steadily gaining in their understanding of BDNF and how it affects brain plasticity and regeneration. It is already established that hyperbaric oxygen therapy can support brain regenerative effects. A new study, published in 2017, continues to support this concept. Researchers used a lower pressure of 1.5 ATA along with 2.0 ATA, and found that both pressures were able to cause significant increases in BDNF in just 3 to 5 days of consecutive HBOT sessions.
“Both, the genetically modified NIH3T3/BDNF and native NIH3T3 fibroblasts, showed a highly significant increased proliferation after five days of HBOT in comparison to normoxic controls. “
A study published in 2012 in “Cell Stress and Chaperones, A Comprehensive Journal of Stress Biology and Medicine” focused on the effects of HBOT preconditioning and its protective properties against Ultraviolet-A (UV-A) induced skin damage. Three groups of hairless mice were exposed to UV-A, three days a week for 22 weeks, with two of the groups receiving HBOT pretreatment either two or four times a week. UV-A exposure amplified skin cell death, signifying elevated levels of skin damage. Pretreatment with HBOT substantially reduced UV-A induced cell death. In addition, HBOT pretreatment prevented skin creasing and maintained skin elasticity.
The following research highlights the benefit of hyperbaric oxygen therapy for anti-aging.
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